What chromosome has been determined to carry the gene for Marfan syndrome?

The answer is probably on a "Human Chromosome Map".

I'm looking for more than just chromosome 15.

Thanks a lot!

"Marfan syndrome has been linked to a defect in the FBN1 gene on chromosome 15,[6] which encodes a glycoprotein called fibrillin-1. Fibrillin is essential for the formation of the elastic fibers found in connective tissue, as it provides the scaffolding for tropoelastin.[3] Elastic fibers are found throughout the body but are particularly abundant in the aorta, ligaments and the ciliary zonules of the eye, consequently these areas are among the worst affected. Without the structural support provided by fibrillin many connective tissues are weakened, which can have severe consequences for support and stability."

Each parent with the condition has a 50% chance of passing it on to a child due to its autosomal dominant nature. Most individuals with Marfan syndrome have another affected family member, but approximately 15-30% of all cases are due to de novo genetic mutations鈥?such spontaneous mutations occur in about 1 in 20 000 births. Marfan syndrome is also an example of dominant negative mutation and haploinsufficiency. It is associated with incomplete penetrance, therefore not all persons carrying the mutation develop the disease.
Marfan syndrome has been linked to a defect in the FBN1 gene on chromosome 15,which encodes a glycoprotein called fibrillin-1. Fibrillin is essential for the formation of the elastic fibers found in connective tissue, as it provides the scaffolding for tropoelastin.Elastic fibers are found throughout the body but are particularly abundant in the aorta, ligaments and the ciliary zonules of the eye, consequently these areas are among the worst affected. Without the structural support provided by fibrillin many connective tissues are weakened, which can have severe consequences for support and stability.
A related disease has been found in mice, and the study of mouse fibrillin synthesis and secretion, and connective tissue formation, has begun to further our understanding of Marfan syndrome in humans. It has been found that simply reducing the level of normal fibrillin-1 causes a Marfan-related disease in mice.

Transforming growth factor beta (TGF尾) plays an important role in Marfan syndrome. Fibrillin-1 binds TGF尾 and inactivates it. In Marfan syndrome, reduced levels of fibrillin-1 allow activated TGF尾 to damage the lungs and heart. A defect in the gene TGF尾R2 on chromosome 3, a receptor protein of TGF尾, has also been related to Marfan syndrome. Marfan syndrome can often be confused with Loeys-Dietz syndrome, a similar connective tissue disorder resulting from mutations in the TGF尾 receptor genes TGF尾R1 and TGF尾R2.