What is the difference between insulin resistance and diabetes?

i am insulin resistant

Diabetes is a disease in which the body does not produce or properly use insulin. Insulin is a hormone produced by the pancreas that allows glucose to enter the cells where it will be turned into energy.

Insulin resistance -means you are a patient of Type 2 diabetes.
Insulin resistance is the condition in which normal amounts of insulin are inadequate to produce a normal insulin response from fat, muscle and liver cells. Insulin resistance in fat cells results in hydrolysis of stored triglycerides, which elevates free fatty acids in the blood plasma. Insulin resistance in muscle reduces glucose uptake whereas insulin resistance in liver reduces glucose storage, with both effects serving to elevate blood glucose. High plasma levels of insulin and glucose due to insulin resistance often leads to the metabolic syndrome and type 2 diabetes.
http://en.wikipedia.org/wiki/Insulin_res...
http://syndromex.stanford.edu/InsulinRes...
http://www.drmirkin.com/diabetes/D222.ht...

In Type 1 diabetes, the body stops making insulin or makes only a small amount. Since insulin cannot be given by mouth, it is given by daily injections.

Type 1 diabetes accounts for 5 to 10 percent of diabetes; it usually occurs in people under age 30, but it can occur at any age.

In Type 2 diabetes, the body either doesn't make enough insulin, or is unable to use the insulin properly (which is called insulin resistance), or both.

Some people with Type 2 diabetes are controlled by diet and exercise, while most either take diabetes pills, or insulin, or a combination of pills and insulin.

Type 2 diabetes accounts for 90 to 95 percent of diabetes, and usually occurs after age 40; however, now it is also occurring more often in children and teens.

For more basic information about diabetes, you might want to check out the American Diabetes Association's web site at:
http://www.diabetes.org

Exercise-induced reversal of insulin resistance in obese elderly is associated with reduced visceral fat.
鈥'Leary VB,
鈥archetti CM,
鈥rishnan RK,
鈥tetzer BP,
鈥onzalez F,
鈥irwan JP.
Schwartz Center for Metabolism and Nutrition, Case Western Reserve University School of Medicine, Cleveland, OH 44109-1998, USA.
Exercise improves glucose metabolism and delays the onset and/or reverses insulin resistance in the elderly by an unknown mechanism. In the present study, we examined the effects of exercise training on glucose metabolism, abdominal adiposity, and adipocytokines in obese elderly. Sixteen obese men and women (age = 63 +/- 1 yr, body mass index = 33.2 +/- 1.4 kg/m2) participated in a 12-wk supervised exercise program (5 days/wk, 60 min/day, treadmill/cycle ergometry at 85% of heart rate maximum). Visceral fat (VF), subcutaneous fat, and total abdominal fat were measured by computed tomography. Fat mass and fat-free mass were assessed by hydrostatic weighing. An oral glucose tolerance test was used to determine changes in insulin resistance. Exercise training increased maximal oxygen consumption (21.3 +/- 0.8 vs. 24.3 +/- 1.0 ml.kg(-1).min(-1), P < 0.0001), decreased body weight (P < 0.0001) and fat mass (P < 0.001), while fat-free mass was not altered (P > 0.05). VF (176 +/- 20 vs. 136 +/- 17 cm2, P < 0.0001), subcutaneous fat (351 +/- 34 vs. 305 +/- 28 cm2, P < 0.03), and total abdominal fat (525 +/- 40 vs. 443 +/- 34 cm2, P < 0.003) were reduced through training. Circulating leptin was lower (P < 0.003) after training, but total adiponectin and tumor necrosis factor-alpha remained unchanged. Insulin resistance was reversed by exercise (40.1 +/- 7.7 vs. 27.6 +/- 5.6 units, P < 0.01) and correlated with changes in VF (r = 0.66, P < 0.01) and maximal oxygen consumption (r = -0.48, P < 0.05) but not adipocytokines. VF loss after aerobic exercise training improves glucose metabolism and is associated with the reversal of insulin resistance in older obese men and women.
PMID: 16373444 [PubMed - indexed for MEDLINE]

hi, it is when your cell have no receptor to insulin , so they don't act when there is a glucose in the blood , which then cause DM.