Why does a person develop anasarca (generalized edema)? The patient is a known CVA, known Diabetic (mellitus)?

The patient is a known CVA, known Diabetic (mellitus) and has Aspiration Pneumonia... she developed generalized edema eventhough her kidneys are properly working and is excreting adequate amount of urine... how does glucose play a role in edema?

please help i need to answer this for my case presentation! thanks!

Glucose doesn't.

She could have heart failure, or oedema from immoblitiy, or generalised vascular disease (increased permeability).

If the patient has developed sepsis from the aspiration pneumonia, she could have developed a type of toxic shock syndrome, where the toxins from the sepsis make the capillaries abnormally permeable, which will cause anasarca.

Urine output is a poor indicator of renal function, so you need to check her U&E's, this will give a clearer indication of renal function.

Her albumin may be abnormally low, this'll lead to anasarca due to a reduction in oncotic pressure within the blood vessel walls.

She may be receiving too much IV fluids.

Good luck!

Gross protein deficiency is one explanation.Hepatic failure is another reason.Heart failure is also possible.But in all these cases urine output is temporarily low..Are you sure you are giving the correct clinical picture?

Causes of peripheral edema are:
high hydrostatic pressure of the veins, leading to poor reabsorption of fluid
venous obstruction, e.g. deep vein thrombosis (typically one-sided)
congestive heart failure
varicose veins
low oncotic pressure
cirrhosis
malnutrition
nephrotic syndrome (renal protein loss)
epidemic dropsy
obstruction of lymph drainage
infection
cancer
fibrosis after surgery
filariasis
inflammation (active secretion of fluid into the interstitial space due to increased membrane permeability by inflammatory mediators):
allergic conditions (e.g. angioedema)
any other form of inflammation (tumor - or swelling - is one of the main characteristics of inflammation)
Most probably, she is suffering from Congestive heart failure. ECG / BUN / Creatinine tests should be conducted.

not really...did you check the albumin level, that could explain; the other possibility is dependent and disuse oedema from the inactivity